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    A different marker to determine arrhythmia potential between elite active cyclists and veterans: T peak T end
    (WILEY-BLACKWELL, 2015) Gormus, Z. Isik Solak; Isik, Bulent; Ciftci, Ozgur; Icli, Abdullah; Togan, Turhan; Aslan, Huseyin; Kutlu, Selim
    [Abstract not Available]
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    Hippocampal cell loss after an anterior and posterior anastomotic vein occlusion model in rats
    (PERGAMON-ELSEVIER SCIENCE LTD, 2011) Aydin, Keramettin; Cokluk, Cengiz; Ayas, Bulent; Onger, Mehmet Emin; Keskin, Ilknur; Ozyasar, Ali; Aslan, Huseyin
    Estimation of the cell number after cortical venous ischemia/infarction induced by anterior and posterior anastomotic veins occlusion in a rat model is very important. Twenty male Sprague-Dawley rats were used in this experiment. Small burr-holes were made over the anterior (the crossing point of the line drawn from the posterior border of the orbital rim and the line drawn along the para-midline to the superior sagittal suture) and posterior (just inferior point of the posterior ending of the zygomatic arch) anastomotic veins. Bipolar coagulation technique and micro-scissor were used to sacrifice the venous vessels after final inspection and description. Specimens were evaluated by histopathological and unbiased stereological methods for microscopic evaluation and volumetric analysis, respectively. Significant cell loss was seen in the pyramidal and granule cells of the cornu ammonis and dentate gyrus of the hippocampus after venous ischemia. Cell loss was also pronounced when seen in the histological examination. The present results suggest that the sacrifice of anterior and posterior anastomotic veins can be used as an experimental rat model in the evaluation of pyramidal and granule cell loss in the hippocampus that often assesses the neural damage inflicted by this intervention. (C) 2011 ISDN. Published by Elsevier Ltd. All rights reserved.
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    A novel marker to determine arrhytmia risk in elite cyclists: T peak T end
    (2017) Isık, Bulent; Gormus, Z Isik Solak; Aslan, Huseyin; Iclı, Abdullah; Kurklu, Galip Bilen; Cıftcı, Ozgur; Togan, Turhan
    In athletes, left ventricular hypertrophy is a physiological response upon routine active sports. If the hypertrophic cardiomyopathy is not diagnosed and treated, it can lead to sudden deaths in athletes. Not so much data is known whether or not it is favorable to use of Tp-e values in order to reflect the arrhythmia risks in asymptomatic elite cyclists. The aim of this study is to examine the risks of regular bike sport on potential arrhythmia in healthy active elite cyclists and veterans by using non-invasive cardiac tests. Study groups were: healthy volunteers (group 1, n28, mean age 35.84.6), active cyclists (group 2, n27, mean age 213.0), veteran cyclists (group 3, n27, mean age 29.57.1 yr). All groups were underwent cardiological examinations, 12 derivation ECG records, transthoracic echocardiography investigations. Tp-e interval, Tp-e dispersion, corrected Tp-e interval, QT interval and Tp-e/QT ratio were calculated from ECG records. Tp-e intervals were measured with Tangent method, corrected Tp-e interval were measured with Bazett formula. Mean Tp-e interval results were as follows according to groups 1, 2, 3; 75.09.3, 88.17.0, 83.28.8 ms, respectively. The Tp-e interval, cTp-e interval, Tp- dispersion, QT interval, and SLI values were significantly higher in active cyclists than the veteran cyclists and the control group (p0.05). Both cyclist groups had also significantly higher Tp-e interval, cTp-e interval, QT interval, Tp-e/QT, QRS time and SLI values than the control group (p0.05). Furthermore, there were significant differences in Tp-e interval, cTp-e interval, Tp-e dispersion, QT interval and SLI values between the active and veteran cyclists (p0.05). There were significant differences in Tp-e dispersion between active cyclists and control group or veteran cyclists (p 0.01). Arrythmia related to Tp-e interval, Tp-e dispersion, corrected Tp-e interval and Tp-e/QT ratio are associated with long-term, intense bike sport training either in active cyclists or veterans. This can also be associated with left ventricular hypertrophy in active cyclists and remnant left ventricular hpertrophy in veterans