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    Accidental displacement of impacted maxillary and mandibular third molars
    (QUINTESSENCE PUBL CO INC, 2004) Durmus, E; Dolanmaz, D; Kucukkolbsi, H; Mutlu, N
    Displacement of impacted third molars is frequently mentioned in oral and maxillofacial surgery textbooks, but rarely reported. However, should this complication arise in general practice, the clinician should not embark on potentially complicated and hazardous surgical procedures to retrieve the displaced tooth. Administration of prophylactic broad spectrum antibiotics and urgent referral to an oral and maxillofacial surgeon, is recommended.
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    Management of early relapse after a sagittal split ramus osteotomy by gradual callus distraction: A case report
    (W B SAUNDERS CO, 2000) Uckan, S; Buchbinder, D; Orhan, M; Mutlu, N
    [Abstract not Available]
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    Non-ossifying fibroma of the mandible: Report of a case
    (CHURCHILL LIVINGSTONE, 1999) Uckan, S; Gurol, M; Mutlu, N; Gungor, S
    [Abstract not Available]
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    T102C polymorphism of the 5-HT2A receptor gene may be associated with temporomandibular dysfunction
    (BLACKWELL MUNKSGAARD, 2004) Mutlu, N; Erdal, ME; Herken, H; Oz, G; Bayazit, YA
    OBJECTIVE: To assess whether a relationship existed between the T102C polymorphism of 5-HT2A receptor gene and temporomandibular dysfunction. METHODS: Sixty-three patients with temporomandibular dysfunction, and 54 healthy volunteer controls were included in the study. Molecular analysis of the T102C polymorphism of the 5-HT2A receptor gene was performed using PCR technique. RESULTS: The C/C genotype was over represented in the patients whereas T/T genotype was over represented in the controls (P < 0.05). The genotype distribution of the patients who had temporomandibular dysfunction was not different than those who did not have temporomandibular dysfunction (P > 0.05). CONCLUSION: The T102C polymorphism may be involved in the etiology of temporomandibular dysfunction. The overrepresentation of the C/C variant of 5-HT2A receptor gene in temporomandibular dysfunction suggests a possible role of the serotonergic system in this disease, particularly at the receptor level.