Yazar "Baltaci, Saltuk Bugra" seçeneğine göre listele

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    The Effect of Pinealectomy and Melatonin Application on Metallothionin, ZnT3 and ZIP2 Levels in Rat Brain Tissue
    (KARGER, 2018) Deniz, Arife; Baltaci, Saltuk Bugra; Baltaci, Abdulkerim Kasim; Mogulkoc, Rasim; Avunduk, Mustafa Cihat
    [Abstract not Available]
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    The Effect of Pinealectomy and Melatonin Supplementation on Serum Melatonin, Nesfatin-1 and Ghrelin Levels in Rats
    (KARGER, 2018) Sonmez, Saadet; Baltaci, Abdulkerim Kasim; Baltaci, Saltuk Bugra; Mogulkoc, Rasim; Menevse, Esma
    [Abstract not Available]
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    The Effect of the Application of Melatonin and Zinc in DMBA-Induced Mammary Carcinoma in Rats on Lipid Peroxidation and Element Metabolism
    (KARGER, 2018) Mutlu, Elif Gulbahce; Baltaci, Saltuk Bugra; Menevse, Esma; Mogulkoc, Rasim; Baltaci, Abdulkerim Kasim
    [Abstract not Available]
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    Levels of Plasma NPY, Leptin and Nesfatin-1, and Their Relation to Zinc in Children with Obese and Metabolic Syndrome
    (KARGER, 2018) Aydogdu, Adnan; Baltaci, Abdulkerim Kasim; Menevse, Esma; Baltaci, Saltuk Bugra; Mogulkoc, Rasim; Erdem, Said Sami; Buyukinan, Muammer
    [Abstract not Available]
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    Melatonin has a protective effect against lipid peroxidation in the bone tissue of diabetic rats subjected to acute swimming exercise
    (WALTER DE GRUYTER GMBH, 2018) Bicer, Mursel; Baltaci, Saltuk Bugra; Patlar, Suleyman; Mogulkoc, Rasim; Baltaci, Abdulkerim Kasim
    Aim: The present study aimed to examine the effects of melatonin supplementation on lipid peroxidation in the bone tissue of diabetic rats subjected to acute swimming exercise. Methods: The study was conducted on 80 Sprague-Dawley type adult male rats which were equally allocated to eight groups: group 1, general control; group 2, melatonin-supplemented control; group 3, melatonin-supplemented diabetic control; group 4, swimming control; group 5, melatonin-supplemented swimming; group 6, melatonin-supplemented diabetic swimming; group 7, diabetic swimming; group 8, diabetic control. In order to induce diabetes, the animals were subcutaneously injected with 40 mg/kg streptozotocin (STZ). The animals were supplemented with 3 mg/kg/day melatonin intraperitoneally (IP) for 4 weeks. At the end of the study, the animals were decapitated to collect bone tissue samples which were examined to find out the malondialdehyde (MDA) (nmol/g/protein) and glutathione (GSH) (mg/dL/g protein) levels. Results: The highest MDA values in the bone tissue were found in groups 7 and 8. MDA levels in the bone tissue in groups 3 and 6 were lower than the levels in groups 7 and 8, but higher than those in all other groups. Groups 3, 5 and 6 had the highest bone tissue GSH values. On the other hand, the lowest GSH level was established in groups 7 and 8. Conclusion: The results of the present study indicated that the cell damage caused by acute swimming exercise and diabetes in the bone tissue could be prevented by melatonin supplementation.
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    Molecular Mechanisms of Early and Late LTP
    (SPRINGER/PLENUM PUBLISHERS, 2019) Baltaci, Saltuk Bugra; Mogulkoc, Rasim; Baltaci, Abdulkerim Kasim
    LTP is the most intensively studied cellular model of the memory and generally divided at least two distinct phases as early and late. E-LTP requires activation of CaMKII that initiates biochemical events and trafficking of proteins, which eventually potentiate synaptic transmission, and is independent of de novo protein synthesis. In contrast, L-LTP requires gene expression and local protein synthesis regulated via TrkB receptor- and functional prions CPEB2-3-mediated translation. Maintenance of LTP for longer periods depends on constitutively active PKM. Throughout this review, current knowledge about early and late phases of LTP will be reviewed.
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    Resveratrol and exercise
    (SPANDIDOS PUBL LTD, 2016) Baltaci, Saltuk Bugra; Mogulkoc, Rasim; Baltaci, Abdulkerim Kasim
    Although it is recommended for a healthy lifestyle, moderate exercise is known to lead to oxidative stress, inflammation and muscle injury. Hence there are efforts to develop dietary strategies to counter the oxidative stress caused by physical activity. Recently, there has been an interest in the capability of resveratrol (RES) to modulate physical performance and prevent oxidative stress. Despite the inconsistency among reports regarding the topic, it has been suggested that RES delays fatigue by hindering lipid peroxidation. It is hypothesized that RES administration produces favorable effects on hepatic cell rejuvenation, exerts a regulatory effect on glucose metabolism, and preserves liver glycogen reserves that are diminished during physical activity. Consequently, there is a growing interest in the association between RES and exercise. The aim of the current review is to interpret the association between RES and exercise.