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    Bacteremia Following Exchange Transfusion in Infants
    (2003) Atabek M.E.; Koc H.; Pirgon O.; Erkul I.
    The hypothesis is that transient bacteremia following exchange transfusion in infants may occur; however, it is not associated with clinical evidence of disease. Therefore, the objective of this study is to evaluate the significance of this bacteremia in terms of clinical evidence of infection. We studied 155 patients with jaundice requiring an immediate exchange transfusion (ET). Blood cultures were taken before, soon after completion of the procedure and at 24h later respectively. The clinical course of each patient after the procedure was monitored in terms of evidence of sepsis for minimum of five days. One hundred twenty of the 155 patients underwent a single ET whereas 35 patients required a second ET within 8 to 12 hours. Blood cultures obtained before ET (preexchange specimen) and at 24h later were sterile in all patients. Eighteen (11.6%) patients had positive blood cultures soon after completion of the procedure. Eleven of 120 (9.1%) patients with a single ET had positive blood cultures. Seven of 35 (20%) patients with the second ETs had positive blood cultures. There was a significant statistical difference in terms of bacteremia between the group of single ET and the group of second ETs. (p<0.001). The incidence of requirement for clinical infection was 1 out of 18 (5.5%) infants who developed bacteremia. Bacteremia resolved spontaneously in the majority of infants. However 1 in 18 (5.5%) of the bacteremic infants did develop symptoms and required treatment.
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    High dose intravenous methylprednisolone in TAR syndrome
    (1994) Oran B.; Caliskan U.; Oran I.; Tastekin A.; Yilmaz H.; Yasar N.C.; Erkul I.
    [Abstract not Available]
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    Prevalence of hepatitis A, B, C and E virus in adolescents with type-1 diabetes mellitus
    (Freund Publishing House Ltd, 2003) Atabek M.E.; Kart H.; Erkul I.
    The hypothesis for this study was that hepatitis virus infection could be associated with diabetes, because of the high frequency of injections. In this study, we aimed to determine the prevalence of hepatitis A, B, C and E viruses in type-1 diabetes mellitus. Methods: Sixty-three patients with the diagnosis of insulin-dependent diabetes mellitus and 63 healthy controls were included in this study. Serological markers of four different types of hepatitis (Anti-HAV IgM, total anti-HAV, HbsAg, anti-HBs, total anti-HBc, antiHBc IgM, anti-HCV and anti-HEV) were studied in all cases. None of the patients had a history of previous icterus or other signs of hepatitis, had received blood transfusions, or were on hemodialysis. Results: There was no difference between the patients and controls with respect to hepatitis A, B, C and E virus serology. The rate of seropositivity of patients within a month of the diagnosis was smaller than those of the patients whose diagnosis were older than one month, but the difference was not found to be statistically significant (p>0,05). Moreover, hepatitis virus infections did not seem to be related to duration of disease, nor age of onset, ketoacidosis, HbA1c, and insulin regimen. All of the sera were negative for anti-HCV in both patients and controls. Conclusion: Serological evidence of previous HAV, HBV, HCV and HEV infections was not significantly different between type-1 diabetes patients and healthy controls. Hepatitis virus infection was not associated with diabetes in spite of the high frequency of injections.