Use of chest circumference signal as an input to models of Respiration-HRV interaction

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dc.contributor.authorYildiz, Metin
dc.contributor.authorIder, Y. Ziya
dc.date.accessioned2020-03-26T17:18:45Z
dc.date.available2020-03-26T17:18:45Z
dc.date.issued2007
dc.departmentSelçuk Üniversitesien_US
dc.descriptionWorld Congress on Medical Physics and Biomedical Engineering -- AUG 27-SEP 01, 2006 -- Seoul, SOUTH KOREAen_US
dc.description.abstractContributions of respiration to the genesis of Heart Rate Variability are well known. Respiration not only generates the HF (High Frequency) component of the power spectrum of HRV, called respiratory sinus arrhythmia, but also contributes to the formation of the LF (Low Frequency) peak. Therefore it is necessary that respiration is also recorded during an HRV test so that LF/HF ratio, which is the most important parameter extracted from HRV for assessing sympathovagal balance, is more properly evaluated. LF components of lung volume (V-L), intrathoracic pressure (P-T), and abdominal pressure (P-A), are more significant especially when respiration is slightly irregular, and may directly contribute to HRV LF power. Models developed to study these interactions need respiratory signal inputs. Measurements of V-L, P-T, and P-A in a clinical setting are either not possible or induce stress in the patient and thus alter HRV. On the other hand chest and abdominal circumference signals (C-T and C-A respectively) are easier to acquire without inducing stress. It is shown that by appropriately scaling and offsetting C-T and C-A one obtains signals representative of P-T and P-A which then can be used as inputs to the models. V-L can also be derived from P-T, through a linear empirical formula published elsewhere. C-T, C-A, and V-L from 5 volunteers are recorded together with ECG. It is shown that C-T is linearly related to V-L, and hence to P-T. The derived P-T and P-A signals are then applied to a Respiration-Cardiovascular System model and HRV is derived. It is observed that model derived and real HRVs are temporally well correlated. It is also shown that in the presence of increased LF power of the circumference signals HRV LF power also increases.en_US
dc.description.sponsorshipIUPSEM, IFMBE, IOMP, KSMP, KSMBE, AAPM, AFOMP, BMES, EFOMP, IAEA, AIMBE, IEEE EMB, WHO, SIEMENS, TomoTherapy, Philips, iba, Varian, CIVCO, Elekta, Samsungen_US
dc.identifier.endpage+en_US
dc.identifier.isbn978-3-540-36839-7
dc.identifier.issn1680-0737en_US
dc.identifier.startpage3445en_US
dc.identifier.urihttps://hdl.handle.net/20.500.12395/21728
dc.identifier.volume14en_US
dc.identifier.wosWOS:000260855901354en_US
dc.identifier.wosqualityN/Aen_US
dc.indekslendigikaynakWeb of Scienceen_US
dc.language.isoenen_US
dc.publisherSPRINGER-VERLAG BERLINen_US
dc.relation.ispartofWORLD CONGRESS ON MEDICAL PHYSICS AND BIOMEDICAL ENGINEERING 2006, VOL 14, PTS 1-6en_US
dc.relation.ispartofseriesIFMBE Proceedings
dc.relation.publicationcategoryKonferans Öğesi - Uluslararası - Kurum Öğretim Elemanıen_US
dc.rightsinfo:eu-repo/semantics/closedAccessen_US
dc.selcuk20240510_oaigen_US
dc.subjectHeart rate variabilityen_US
dc.subjectRespirationen_US
dc.subjectPhysiological modelingen_US
dc.subjectChest circumferenceen_US
dc.subjectSimulationen_US
dc.titleUse of chest circumference signal as an input to models of Respiration-HRV interactionen_US
dc.typeConference Objecten_US

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