Gallbladder Motor Function in Patients with Different Thyroid Hormone Status

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dc.contributor.authorCakir, M.
dc.contributor.authorKayacetin, E.
dc.contributor.authorToy, H.
dc.contributor.authorBozkurt, S.
dc.date.accessioned2020-03-26T17:38:45Z
dc.date.available2020-03-26T17:38:45Z
dc.date.issued2009
dc.departmentSelçuk Üniversitesien_US
dc.description.abstractHypothyroidism and hyperthyroidism are known to affect cholesterol metabolism, bile acid synthesis and bile content. There are previous Studies reporting increased prevalence of gallstone and common bile duct stone formation in hypothyroidism. The aim Of this Study was to compare gallbladder (GB) motor function between euthyroid, hypothyroid and hyperthyroid Subjects by conventional ultrasonography. Eighteen euthyroid, 14 hypothyroid and 20 hyperthyroid, age, sex and body mass index matched subjects were included in the study. Etiology of hypothyroidism comprised of thyroidectomy, Hashimoto's thyroiditis and previous radioactive iodine therapy. Hyperthyroid group included patients with toxic nodular goitre and Graves' disease. Patients who have diseases or are under drug treatment known to affect biliary function were not included in the study. Fasting and post-stimulus GB volumes were measured by real-time conventional ultrasonography and computer unit (Aloka UST-979, 3.5 mHz electronic convex probe, Japan). No significant differences were noted for fasting, post-stimulus GB volumes and GB ejection fraction between euthyroid, hypothyroid and hyperthyroid subjects. Measuring GB motility accurately is not straightforward because besides being not static during fasting, after a meal, intermittent emptying and refilling of GB occurs. Ultrasonography is affected by GB filling and measures "net" GB emptying. Although cumbersome, additional evaluation with cholescintigraphy which detects "absolute" GB emptying and is not affected by GB filling may provide complete assessment of motor function and may be more informative in evaluating subjects with different thyroid hormone status.en_US
dc.identifier.doi10.1055/s-0029-1202850en_US
dc.identifier.endpage399en_US
dc.identifier.issn0947-7349en_US
dc.identifier.issue8en_US
dc.identifier.pmid19373757en_US
dc.identifier.scopusqualityQ2en_US
dc.identifier.startpage395en_US
dc.identifier.urihttps://dx.doi.org/10.1055/s-0029-1202850
dc.identifier.urihttps://hdl.handle.net/20.500.12395/23565
dc.identifier.volume117en_US
dc.identifier.wosWOS:000270680100006en_US
dc.identifier.wosqualityQ4en_US
dc.indekslendigikaynakWeb of Scienceen_US
dc.indekslendigikaynakScopusen_US
dc.indekslendigikaynakPubMeden_US
dc.language.isoenen_US
dc.publisherJOHANN AMBROSIUS BARTH VERLAG MEDIZINVERLAGE HEIDELBERG GMBHen_US
dc.relation.ispartofEXPERIMENTAL AND CLINICAL ENDOCRINOLOGY & DIABETESen_US
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanıen_US
dc.rightsinfo:eu-repo/semantics/closedAccessen_US
dc.selcuk20240510_oaigen_US
dc.subjectgallbladder motor functionen_US
dc.subjecthypothyroidismen_US
dc.subjecthyperthyroidismen_US
dc.titleGallbladder Motor Function in Patients with Different Thyroid Hormone Statusen_US
dc.typeArticleen_US

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