The effect of diabetes mellitus on the P-wave dispersion

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dc.contributor.authorYazıcı, Mehmet
dc.contributor.authorÖzdemir, Kurtuluş
dc.contributor.authorAltunkeser, Bülent Behlül
dc.contributor.authorKayrak, Mehmet
dc.contributor.authorDüzenli, M. Akif
dc.contributor.authorVatankulu, M. Akif
dc.contributor.authorSoylu, Ahmet
dc.date.accessioned2020-03-26T17:18:26Z
dc.date.available2020-03-26T17:18:26Z
dc.date.issued2007
dc.departmentSelçuk Üniversitesien_US
dc.description.abstractBackground P-wave dispersion (PD), a measure of heterogeneity of atrial refractoriness, is defined as the difference between the minimum (P min) and maximum P-wave (P max) durations on standard 12-lead electrocardiography (ECG). Increase in PD shows the intra-atrial and inter-atrial non-uniform conduction. In the present study the evaluation of the effect of diabetes mellitus (DM) on PD in patients without coronary artery disease and hypertension was carried out. Methods and Results Seventy-six diabetic patients who had no coronary artery disease or hypertension (group 1; mean age 48 +/- 9) and 40 healthy volunteer individuals (group2; mean age 46 +/- 13) were enrolled in the study. After obtaining 12-lead surface ECG of all cases, P max and P min P-wave durations were measured and the differences between them were taken as PD (PD=P max-P min). Left atrium diameter, left ventricular end systolic and end diastolic diameters were measured and left ventricular ejection fraction was determined by echocardiography. Pulse wave mitral flow velocities were measured from the apical 4-chamber view. Mitral early diastolic velocity (E), late diastolic velocity (A), E/A, E deceleration time and isovolumetric relaxation time were determined. In comparison of the 2 groups there was no statistically significant difference among age, sex, systolic and diastolic blood pressure, resting heart rate and body mass index of the cases. Although PD and P max were significantly higher in diabetic patients, there was no difference between P min values (33 12 vs 28 +/- 10, p=0.02; 99 +/- 12 vs 93 +/- 10, p=0.011; 66 +/- 9 vs 65 +/- 10, p=NS; respectively). Conclusions DM might increase PD, even without ischemia, hypertension and left ventricular hypertrophy.en_US
dc.identifier.doi10.1253/circj.71.880en_US
dc.identifier.endpage883en_US
dc.identifier.issn1346-9843en_US
dc.identifier.issn1347-4820en_US
dc.identifier.issue6en_US
dc.identifier.pmid17526984en_US
dc.identifier.scopusqualityQ1en_US
dc.identifier.startpage880en_US
dc.identifier.urihttps://dx.doi.org/10.1253/circj.71.880
dc.identifier.urihttps://hdl.handle.net/20.500.12395/21664
dc.identifier.volume71en_US
dc.identifier.wosWOS:000246882800012en_US
dc.identifier.wosqualityQ2en_US
dc.indekslendigikaynakWeb of Scienceen_US
dc.indekslendigikaynakScopusen_US
dc.indekslendigikaynakPubMeden_US
dc.language.isoenen_US
dc.publisherJAPANESE CIRCULATION SOCen_US
dc.relation.ispartofCIRCULATION JOURNALen_US
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanıen_US
dc.rightsinfo:eu-repo/semantics/openAccessen_US
dc.selcuk20240510_oaigen_US
dc.subjectdiabetes mellitusen_US
dc.subjectP-wave dispersionen_US
dc.titleThe effect of diabetes mellitus on the P-wave dispersionen_US
dc.typeArticleen_US

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