First case report of neurobrucellosis associated with hydrocephalus

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dc.contributor.authorGuney, Figen
dc.contributor.authorGumus, Haluk
dc.contributor.authorOgmegul, Aysegul
dc.contributor.authorKandemir, Bahar
dc.contributor.authorEmlik, Dilek
dc.contributor.authorArslan, Ugur
dc.contributor.authorTuncer, Inci
dc.date.accessioned2020-03-26T17:26:55Z
dc.date.available2020-03-26T17:26:55Z
dc.date.issued2008
dc.departmentSelçuk Üniversitesien_US
dc.description.abstractBrucellosis is a common zoonosis in many parts of the world, including Mediterranean and Middle Eastern countries. The disease is primarily related to occupations at risk, such as veterinarians, farmers, laboratory technicians, abattoir workers, and others working with animals and their products. Neurologic complications of brucellosis are quite rare, ranging from 1.7 to 10% of those infected. To date, no cases of neurobrucellosis with hydrocephalus have been reported. A 38-year-old right-handed farmer complained of headaches, nausea, vomiting, gait disturbance, and sweating for 2 days. He also complained of bilateral hearing loss of 4 months duration. On neurologic examination, dysmmetry, dysdiadochokinesis, ataxia on the left, and bilateral sensorineural hearing loss existed. On cranial MRI, a communicating hydrocephalus was noted. Because the patient consumed fresh sheep cheese and was a farmer, brucellosis was considered in the differential diagnosis. Brucella agglutination was positive with a 1/320 titer in the blood and a 1/80 titer in the cerebrospinal fluid. Ceftriaxone, doxycycline, and rifampicin were administered and by the fourth week of treatment, the ataxia was markedly improved, and the patient was able to walk without support. His cranial MRI demonstrated a total regression of the hydrocephalus. As a result, we suggest that neurobrucellosis should be considered in patients with hydrocephalus, especially if they live in an endemic area for brucellosis, even in the absence of other systemic signs. (c) 2008 Elsevier B.V. All rights reserved.en_US
dc.identifier.doi10.1016/j.clineuro.2008.04.002en_US
dc.identifier.endpage742en_US
dc.identifier.issn0303-8467en_US
dc.identifier.issn1872-6968en_US
dc.identifier.issue7en_US
dc.identifier.pmid18499339en_US
dc.identifier.scopusqualityQ2en_US
dc.identifier.startpage739en_US
dc.identifier.urihttps://dx.doi.org/10.1016/j.clineuro.2008.04.002
dc.identifier.urihttps://hdl.handle.net/20.500.12395/22417
dc.identifier.volume110en_US
dc.identifier.wosWOS:000258368100018en_US
dc.identifier.wosqualityQ3en_US
dc.indekslendigikaynakWeb of Scienceen_US
dc.indekslendigikaynakScopusen_US
dc.indekslendigikaynakPubMeden_US
dc.language.isoenen_US
dc.publisherELSEVIER SCIENCE BVen_US
dc.relation.ispartofCLINICAL NEUROLOGY AND NEUROSURGERYen_US
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanıen_US
dc.rightsinfo:eu-repo/semantics/closedAccessen_US
dc.selcuk20240510_oaigen_US
dc.subjectneurobrucellosisen_US
dc.subjecthydrocephalusen_US
dc.subjectmeningitisen_US
dc.titleFirst case report of neurobrucellosis associated with hydrocephalusen_US
dc.typeArticleen_US

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