The role of hyperbaric oxygen in the management of subarachnoid hemorrhage

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dc.contributor.authorKocaogullar, Y
dc.contributor.authorUstun, ME
dc.contributor.authorAvci, E
dc.contributor.authorKarabacakoglu, A
dc.contributor.authorFossett, D
dc.date.accessioned2020-03-26T16:55:35Z
dc.date.available2020-03-26T16:55:35Z
dc.date.issued2004
dc.departmentSelçuk Üniversitesien_US
dc.description.abstractObjective: To determine the role of hyperoxic and hyperbaric therapy following experimental subarachnoid hemorrhage (SAH). Design: Prospective, randomized, controlled animal study. Subjects: Thirty male Wistar rats. Interventions: Thirty rats were assessed for an initial neurologic status as double-blinded by two different neurosurgeons using a neurologic severity score (NSS) and then underwent an initial angiographic examination. Two days later, 0.3 ml of homologous blood was injected into the cisterna magna to produce a SAH-induced cerebral vasospasm. The NSS and angiographic examination were then repeated. The rats having no spasm or a spasm under 50% (n=8) and 50% or over 50% (n=22) were grouped separately, as groups 1 and 2, respectively. The rats having 50% or more spasm were further divided randomly into group 2A and 2B. The rats in groups 1 and 2A (n= 11) underwent a 60-min course of 100% oxygen at the atmospheric pressure 1 atmosphere absolute (ata), and group 2B (n= 11) received 100% oxygen at 3 ata for I h. Neurologic assessment was repeated on the next day and 7 days later. Measurements and main results: The animals having no spasm or less than 50% spasm had a better NSS and outcome when compared with the animals having 50% or more spasm. But the animals with 50% or more spasm which underwent hyperbaric therapy were shown to have a better outcome compared to the animals having hyperoxic therapy. Conclusion: Exposure to hyperbaric oxygen therapy seemed to accelerate the recovery of neurologic deficits secondary to experimental SAH.en_US
dc.identifier.doi10.1007/s00134-003-1916-7en_US
dc.identifier.endpage146en_US
dc.identifier.issn0342-4642en_US
dc.identifier.issn1432-1238en_US
dc.identifier.issue1en_US
dc.identifier.pmid12904851en_US
dc.identifier.scopusqualityQ1en_US
dc.identifier.startpage141en_US
dc.identifier.urihttps://dx.doi.org/10.1007/s00134-003-1916-7
dc.identifier.urihttps://hdl.handle.net/20.500.12395/19258
dc.identifier.volume30en_US
dc.identifier.wosWOS:000189304000022en_US
dc.identifier.wosqualityQ2en_US
dc.indekslendigikaynakWeb of Scienceen_US
dc.indekslendigikaynakScopusen_US
dc.indekslendigikaynakPubMeden_US
dc.language.isoenen_US
dc.publisherSPRINGERen_US
dc.relation.ispartofINTENSIVE CARE MEDICINEen_US
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanıen_US
dc.rightsinfo:eu-repo/semantics/openAccessen_US
dc.selcuk20240510_oaigen_US
dc.subjectsubarachnoid hemorrhageen_US
dc.subjecthyperbaric oxygenen_US
dc.subjectvasospasmen_US
dc.subjectraten_US
dc.subjectangiographyen_US
dc.titleThe role of hyperbaric oxygen in the management of subarachnoid hemorrhageen_US
dc.typeArticleen_US

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