Pre and Postprandial Total Serum Bile Acid Concentration Following Acute Liver Damage in Dogs

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dc.contributor.authorTurgut, K.
dc.contributor.authorDemir, C.
dc.contributor.authorOk, M.
dc.contributor.authorÇiftçi, K.
dc.date.accessioned2020-03-26T16:25:26Z
dc.date.available2020-03-26T16:25:26Z
dc.date.issued1997
dc.departmentSelçuk Üniversitesien_US
dc.description.abstractThe importance of preprandial and postprandial total bile acids were investigated in dogs with liver damage induced by carbon tetrachloride (CCl4) administration. Six healthy and mature dogs were used. After base-line clinical and biochemical examinations, hepatocellular damage was induced by oral CCl4 administration. Determinations of plasma total protein (TP), albumin (Alb), total and direct bilirubin (TBil, DBil), alanine aminotransferase (ALT) and alkaline phosphatase (ALP) along with histologic ex-amination of the livers 10 days following CCl4 administration were conducted to ensure that hepatic damage was in fact induced by the CCl4 administration. Twelve h fasting preprandial and 2 h postprandial serum total bile acids (PRSBA, POSBA) concentrations were also measured. The median plasma TP, Alb, TBil and DBil concentrations did nor show any difference (P > 0.05) during the study. Plasma ALT activities increased significantly (P < 0.05, P < 0.01) between on the 3rd and 10th days of the experiment. The median serum values of PRSBA and POSBA were significantly different (P < 0.01) on the first day and continued to be different during the experiment. In conclusion, POSBA seemed to be more reliable than PRSBA in the diagnosis of hepatic necrosis.en_US
dc.identifier.citationTurgut, K., Demir, C., Ok, M., Çiftçi, K., (1997). Pre and Postprandial Total Serum Bile Acid Concentration Following Acute Liver Damage in Dogs. Journal of Veterinary Medicine Series a-Physiology Pathology Clinical Medicine, 44(1), 25-29.
dc.identifier.doi10.1111/j.1439-0442.1997.tb01083.xen_US
dc.identifier.endpage29en_US
dc.identifier.issn0931-184Xen_US
dc.identifier.issue1en_US
dc.identifier.pmid9123980en_US
dc.identifier.scopusqualityN/Aen_US
dc.identifier.startpage25en_US
dc.identifier.urihttps://dx.doi.org/10.1111/j.1439-0442.1997.tb01083.x
dc.identifier.urihttps://hdl.handle.net/20.500.12395/16591
dc.identifier.volume44en_US
dc.identifier.wosWOS:A1997WJ53200002en_US
dc.identifier.wosqualityQ3en_US
dc.indekslendigikaynakWeb of Scienceen_US
dc.indekslendigikaynakScopusen_US
dc.indekslendigikaynakPubMeden_US
dc.institutionauthorTurgut, K.
dc.institutionauthorDemir, C.
dc.institutionauthorOk, M.
dc.institutionauthorÇiftçi, K.
dc.language.isoenen_US
dc.publisherBLACKWELL WISSENSCHAFTS-VERLAG GMBHen_US
dc.relation.ispartofJournal of Veterinary Medicine Series a-physiology Pathology Clinical Medicineen_US
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanıen_US
dc.rightsinfo:eu-repo/semantics/openAccessen_US
dc.selcuk20240510_oaigen_US
dc.titlePre and Postprandial Total Serum Bile Acid Concentration Following Acute Liver Damage in Dogsen_US
dc.typeArticleen_US

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